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William Llewellyn - Body Sculpting Bibles Sport Supplement Reference Guide by William Llewellyn 1 book

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William Llewellyn Body Sculpting Bibles Sport Supplement Reference Guide by William Llewellyn 1 book
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The complete reference guide to AAS and performanace enhancing substances.

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William Llewellyns, ANABOLICS, E-Book Edition
Also by this author:
Electronic Books
William Llewellyns
SPORT SUPPLEMENT REFERENCE GUIDE - Kindle Edition
Print Editions
William Llewellyns
SPORT SUPPLEMENT REFERENCE GUIDE
ISBN-13: 978-0-9679304-8-0
ISBN-10: 0-9679304-8-0
Underground Anabolics
ISBN-13: 978-0967930497
ISBN-10: 0967930499
WILLIAM LLEWELLYNS
ANABOLICS
E-Book Edition
DISCLAIMER: This information was gathered from sources including, but not limited to medical journals, pharmaceutical reports, laboratory reports, textbooks, as well as interviews with medical experts, athletes, and steroid distributors. Neither the author nor publisher assumes any liability for the information presented. This book is intended to provide a compendium of information for the reader. None of the information is meant to be applied and is for entertainment purposes only. It is not intended to provide nor replace medical advice. Readers are advised that the substances described in this reference book are to be used only under a physicians care and may be prohibited in certain jurisdictions. Readers should consult with appropriate medical authorities before using any drug, and proper legal authorities on the status of substances described herein. Neither the publisher nor author advocate readers engage in any illegal activities. Copyright 2011 and published by Molecular Nutrition, LLC in Jupiter, FL 33458. All rights reserved.None of the content in this publication may be reproduced, stored in a retrieval system, resold, redistributed, or transmitted in any form or by any means (digital, electronic, mechanical, photocopy, recorded, or otherwise) without the prior written permission of the publisher. William Llewellyns ANABOLICS are trademarks used herein under license. Molecular Nutrition LLC, 5500 Military Trail, Ste. 22-308, Jupiter, FL 33458 www.mnbody.com New Service (HRT-Rx): As longtime advocates of the legitimate medical use of anabolic/androgenic steroids, we have launched HRT-Rx, a national referral service to help patients find progressive HRT/Anti-Aging physicians. Network doctors are the type that regularly work with testosterone medications, and understand the value of hormone replacement and optimization therapies. You can find a physician near you at www.HRT-Rx.com. Note that most offices require you are at least 30 years of age to make an appointment. New Service (HRT-Labs):
We have also partnered with a laboratory that can run extensive blood tests including hormone levels, liver/kidney enzymes, and general health markers. The service is available in all 50 states and can be accessed at www.HRT-Labs.com.
Table of Contents
PART I: ANABOLIC OVERVIEW PART II: PRACTICAL APPLICATION PART III: DRUG PROFILES ANABOLIC/ANDROGENIC STEROIDS (Listed by Common Brand) ANABOLIC AGENTS (NON-STEROID) ANTI-ESTROGENS ANTI-PROLACTIN APPETITE STIMULANTS CARDIOVASCULAR SUPPORT DIURETICS ENDURANCE/ERYTHROPOIETIC DRUGS FAT LOSS AGENTS SYMPATHOMIMETICS FAT LOSS AGENTS THYROID FAT LOSS AGENTS OTHER GROWTH HORMONES & RELATED HYPOGLYCEMICS HYPOTENSIVES LIVER DETOXIFICATION REDUCTASE INHIBITORS TESTOSTERONE STIMULATING DRUGS Drug Profiles Bibliography
Part I
Anabolic Overview

An Introduction to Testosterone

Anabolic steroids are a class of medications that contain a synthetically manufactured form of the hormone testosterone, or a related compound that is derived from (or similar in structure and action to) this hormone. In order to fully grasp how anabolic steroids work, it is, therefore, important to understand the basic functioning of testosterone.
Testosterone is the primary male sex hormone. It is manufactured by the Leydig's cells in the testes at varying amounts throughout a person's life span. The effects of this hormone become most evident during the time of puberty, when an increased output of testosterone will elicit dramatic physiological changes in the male body. This includes the onset of secondary male characteristics such as a deepened voice, body and facial hair growth, increased oil output by the sebaceous glands, development of sexual organs, maturation of sperm, and an increased libido. Indeed the male reproductive system will not function properly if testosterone levels are not significant. All such effects are considered the masculinizing or androgenic properties of this hormone.
Increased testosterone production will also cause growth promoting or anabolic changes in the body, including an enhanced rate of protein synthesis (leading to muscle accumulation). Testosterone is the reason males carry more muscle mass than women, as the two sexes have vastly contrasting amounts of this hormone. More specifically, the adult male body will manufacture between 2.5 and 11mg per day1 while females only produce about 1/4mg. The dominant sex hormone for women is estrogen, which has a significantly different effect on the body. Among other things, a lower androgen and higher estrogen level will cause women to store more body fat, accumulate less muscle tissue, have a shorter stature, and become more apt to bone weakening with age (osteoporosis).
The actual mechanism in which testosterone elicits these changes is somewhat complex. When free in the blood stream, the testosterone molecule is available to interact with various cells in the body. This includes skeletal muscle cells, as well as skin, scalp, kidney, bone, central nervous system, and prostate tissues. Testosterone binds with a cellular target in order to exert its activity, and will, therefore, effect only those body cells that posses the proper hormone receptor site (specifically the androgen receptor). This process can be likened to a lock and key system, with each receptor (lock) only being activated by a particular type of hormone (key). During this interaction, the testosterone molecule will become bound to the intracellular receptor site (located in the cytosol, not on the membrane surface), forming a new receptor complex. This complex (hormone + receptor site) will then migrate to the cell's nucleus, where it will attach to a specific section of the cell's DNA, referred to as the hormone response element. This will activate the transcription of specific genes, which in the case of a skeletal muscle cell will ultimately cause (among other things) an increase in the synthesis of the two primary contractile proteins, actin and myosin (muscular growth). Carbohydrate storage in muscle tissue may be increased due to androgen action as well.
Once this messaging process is completed, the complex will be released, and the receptor and hormone will disassociate. Both are then free to migrate back into the cytosol for further activity. The testosterone molecule is also free to diffuse back into circulation to interact with other cells. The entire receptor cycle, including hormone binding, receptor-hormone complex migration, gene transcription and subsequent return to cytosol is a slow process, taking hours, not minutes, to complete. For example, in studies using a single injection of nandrolone, it is measured to be 4 to 6 hours before free androgen receptors migrate back to the cytosol after activation. It is also suggested that this cycle includes the splitting and formation of new androgen receptors once returned to cytosol, a possible explanation for the many observations that androgens are integral in the formation of their own receptor sites.2
In the kidneys, this same process works to allow androgens to augment erythropoiesis (red blood cell production).3 It is this effect that leads to an increase in red blood cell concentrations, and possibly increased oxygen transport capacity, during anabolic/androgenic steroid therapy. Many athletes mistakenly assume that oxymetholone and boldenone are unique in this ability, due to specific uses or mentions of this effect in drug literature. In fact, stimulation of erythropoiesis occurs with nearly all anabolic/androgenic steroids, as this effect is simply tied with activation of the androgen receptor in kidney cells. The only real exceptions might be compounds such as dihydrotestosterone and some of its derivatives,4 which are rapidly broken down upon interaction with the 3alpha-hydroxysteroid dehydrogenase enzymes (kidney tissue has a similar enzyme distribution to muscle tissue, see section), and therefore display low activity in these tissues.
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