Unknown - THE PERFECT DIABETES COOKBOOK The Step by Step Way to Eat the Foods You Love Including Easy And Healthy Recipes for Diabetes Food Hub nodrm

Contents Copyright 2020.EMILY ROBERT. All rights reserved. No part of this publication may be reproduced, distributed, or transmitted in any form or by any means, including photocopying, recording, or other electronic or mechanical methods, without the prior written permission of the publisher, except in the case of brief quotations embodied in critical reviews and certain other noncommercial uses permitted by copyright law. INTRODUCTION Diabetes Mlltu ("dbt" fr short) a ru d that ur whn your body h dffult rrl regulating the amount of dlvd ugr (glucose) in ur bld stream. It unrelated t a mlrl nmd drdr "Diabetes Insipidus" whh nvlv kdn-rltd fluid retention rblm. In rdr t undrtnd dbt, t necessary to first understand the role glu l wth rgrd to th bd, and wht n hn when rgultn f glu fl nd blood ugr levels bm dangerously lw r hgh. Th tu and cells that mk u th humn body are lvng things, and require fd t t alive.

Th fd ll eat is a t f ugr called glu. Fixed n l th are, the body's cells r completely dndnt n th bld trm n whh they r bthd t brng glu to thm. Wthut access to adequate glu, the body's ll have nothing to fuel themselves wth nd n d. Humn bng t food, nt glu. Human foods gt nvrtd nt glu as a rt f th normal dgtn process. On nvrtd, glu ntr th blood stream, causing th lvl f dissolved glu nd th bld to r.

Th bld trm thn rr th dlvd glucose t th various tu nd cells of th bd. Thugh glucose m be available n th bld, nrb ll are nt bl t access tht glucose without th aid f a hml hormone called nuln. Inuln acts a k t open th ll, llwng them t receive nd utlz available glucose. Cells absorb glu from th bld n the rn f nuln, and bld sugar lvl drop as sugar lv the blood nd ntr th cells. Insulin n b thught f a bridge for glu btwn the bld stream and ll. It mrtnt to undrtnd when lvl f nuln nr, lvl of ugr n th bld decrease (bu the ugr g nt the cells t be used fr nrg).

Th body designed to regulate nd buffr the amount of glu dlvd n the blood t mntn a steady ul to mt ll needs. The pancreas, n f ur body's mn rgn, rdu, tr and releases insulin into th bld trm t brng glu lvl bk dwn. Th concentration of glu vlbl in th blood trm t any given moment dndnt on th munt nd t of fd tht people eat. Rfnd rbhdrt, candy nd wt r t brk down nt glu. Correspondingly, bld glu levels rise rdl after uh fd have been tn. In ntrt, bld sugars r grdull nd slowly ftr tng mr mlx, unrefined rbhdrt (tml, l, baked potatoes, t.) whh rur more digestive steps tk place before glucose can b yielded.

Fd with rdl rng blood glu nntrtn, th bd must react ukl b rlng lrg munt f nuln ll t n or rk a dngru condition called Hrglm (hgh bld ugr) which wll b described blw. Th nflux f insulin nbl ll t utilize glu, nd glucose concentrations dr. While glu lvl can r nd fall rdl, insulin levels change much more lwl. Whn a lrg amount of simple ugr tn the bldtrm quickly becomes fldd wth glu. Inuln is rld by th nr n rn t th increased sugar. The glucose rdl enters the cells but the hgh levels f nuln rmn n th bldtrm for a rd f tm.

This n rult n an overabundance f insulin n th blood, which n trigger feelings f hunger and vn Hypoglycemia (lw bld sugar), nthr ru ndtn. When bld glucose nntrtn rise mr gradually, there l nd fr dramatic compensation. Insulin n b released n a mr ntrlld and safer mnnr which requires th body xrn l strain. Th mr gradual r wll lv u feeling full r ntnt fr a lngr rd f tm. Fr these reasons, t bt for vrll hlth t limit th munt and frequency f wt and rfnd ugr n ur diet. Intd t more mlx sugars uh rw fruit, whl wht brd nd t, and beans.

Th dffrn btwn ml nd complex ugr (carbohydrates) xmlfd b th difference between white (ml) and whl wht (mr mlx) bread. Inuln the rtl k to th ll' ability to u glu. Prblm with nuln rdutn or wth hw nuln recognized by the ll n l u the bd' carefully balanced glu mtblm system to gt ut of control. Whn thr f th problems occur, Dbt dvl, bld sugar lvl surge and crash nd th bd risks bmng dmgd. K ft Th numbr f people wth dbt rose frm 108 million n 1980 to 422 mlln n 2014. Th glbl rvln of dbt* among dult over 18 r f age r frm 4.7% n 1980 t 8.5% in 2014.

Btwn 2000 nd 2016, there was a 5% increase n premature mrtlt frm dbt. Dbt rvln has bn rng mr rapidly in lw nd mddl-nm untr than n hgh-nm untr. Dbt a mjr cause of blndn, kdn flur, hrt ttk, stroke and lwr lmb muttn. In 2016, n estimated 1.6 mlln deaths wr directly caused by dbt. Another 2.2 mlln dth wr ttrbutbl t high blood glucose n 2012. Almt half f ll deaths ttrbutbl t high bld glu ur bfr the g of 70 r.

WHO estimates tht dbt was the vnth ldng u f dth n 2016. A healthy dt, rgulr hl activity, maintaining a nrml bd wght nd avoiding tb u r ways t rvnt r delay the nt f t 2 diabetes. Dbt can be trtd and t nun vdd or delayed with dt, physical tvt, medication and rgulr screening and treatment fr mltn. Overview Diabetes is a hrn disease tht occurs either when the pancreas d not rdu nugh nuln r whn the bd cannot fftvl u th insulin it rdu. Insulin a hormone tht rgult bld sugar. Hrglm, r raised bld ugr, is a common effect f unntrlld dbt and vr time ld t serious damage t many f th bd' tm, ll the nerves and bld vl.

In 2014, 8.5% f adults aged 18 years nd older hd diabetes. In 2016, diabetes w th drt cause f 1.6 million dth nd n 2012 hgh bld glu w the u f nthr 2.2 million dth. Btwn 2000 nd 2016, thr w a 5% nr n rmtur mrtlt frm diabetes. In hgh-nm countries th rmtur mrtlt rate du t diabetes drd frm 2000 to 2010 but thn increased n 2010-2016. In lower-middle-income countries, the rmtur mrtlt rt du t dbt increased r both rd. CHAPTER ONEWht Dbt? Dbt is a disease tht ur when your blood glucose, l lld bld sugar, t hgh. CHAPTER ONEWht Dbt? Dbt is a disease tht ur when your blood glucose, l lld bld sugar, t hgh.

Bld glucose ur main ur f energy and m from the fd u eat. Inuln, a hormone made b the nr, hl glu frm fd get into ur ll t b ud for nrg. Sometimes your bd doesnt mk nughr nnuln r dnt use nuln wll. Glucose thn t in your bld nd dnt rh ur cells. Ovr tm, hvng t muh glucose n ur bld n u hlth problems. Although dbt h n cure, you can take t to mng ur dbt and stay hlth.

Sometimes l ll dbt a tuh f ugr r borderline diabetes. Th trm uggt tht someone dnt rll have diabetes or h a l ru , but vr of diabetes serious. Wht are th dffrnt t fdiabetes? Th mt mmn types f diabetes are t 1, t 2, and gttnl dbt. T 1 dbt If u hv t 1 dbt, ur bd does not mk insulin. Your mmun tm attacks nd dtr th ll n ur nr tht make nuln. T 1 dbt uull dgnd in children nd ung dult, although t n r at any g.

Pl wth t 1 dbt need to tk insulin vr d t t alive. T 2 dbt If u hv type 2 dbt, ur bd does not mk or use nuln wll. You n dvl type 2 dbt t n g, vn durng hldhd. Hwvr, th type f diabetes occurs mt ftn n middle-aged nd ldr people. T 2 is th mt mmn type f dbt. Gttnl dbt Gttnl dbt dvl n m women whn they are rgnnt.

Most f th tm, th t f diabetes g w ftr the bb is born. Hwvr, if youve hd gestational diabetes, you have a grtr hn of dvlng type 2 diabetes later n life. Smtm diabetes dgnd during rgnn is actually t 2 diabetes. Othr t of diabetes Less common t nlud mngn dbt, whh is an nhrtd frm f dbt, nd cystic fibrosis-related dbt Extrnl lnk. Causes of dbt Dffrnt causes r td wth h type f dbt. Type 1 dbt Dtr dnt knw exactly wht causes type 1 diabetes.

For some rn, the immune system mtknl ttk nd dtr insulin-producing beta ll n th nr. Genes m l a rl in m people. It l bl that a vru t off th mmun tm ttk. T 2 dbt Type 2 dbt tm from a mbntn f gnt nd lifestyle ftr. Bng vrwght r b nr your rk too. Carrying xtr wght, ll n ur belly, mk your ll mr rtnt t th effects of nuln n ur blood ugr.

This ndtn runs n fml. Family mmbr hr genes that mk them mr lkl t gt t 2 dbt and t b vrwght. Gestational dbt Gttnl dbt th rult f hrmnl hng durng pregnancy. The lnt produces hrmn tht mk a rgnnt wmn cells less ntv t the effects f nuln. Th can u hgh blood sugar durng rgnn. How does T 2 dbt affect your