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HC-HealthComm (editor) - 12 Endocrinology e-charts: More than 200 images and tables

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12 Endocrinology e-charts: More than 200 images and tables: summary, description and annotation

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The First Edition of 12 Endocrinology e-charts: These Anatomical & Pathological charts were created by Anejo Health Communications.
This reference is an essential addition to every library, whether you are a health professional, student, or interested consumer. Anejo charts show the human body in a format that provides a clear and visual understanding of endocrinology diseases.
This 12 Endocrinology e-charts is ideal for studying human pathologies, patient consultation, or quick reference.
More than 200 images and tables, you may view each image, enlarge it to see every detail.
Table of Contents
Hyperthyroidism e-chart
Glargine e-chart
Obesity e-chart
Signs and Symptoms of Hypothyroidism e-chart
Metabolic syndrome e-chart
Infantile obesity e-chart
Type 2 Diabetes Mellitus e-chart
Diabetes e-chart
Hypothyroidism e-chart
Hyperparathyroidism e-chart
Acromegaly e-chart
Insulin resistance and type 2 diabetes e-chart

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The First Edition of 12 Endocrinology e-charts These Anatomical - photo 1
The First Edition of 12 Endocrinology e-charts These Anatomical - photo 2
The First Edition of 12 Endocrinology e-charts These Anatomical - photo 3
The First Edition of "12 Endocrinology e-charts": These Anatomical & Pathological charts were created by Anejo Health Communications.
This reference is an essential addition to every library, whether you are a health professional, student, or interested consumer. Anejo charts show the human body in a format that provides a clear and visual understanding of endocrinology diseases.
This "12 Endocrinology e-charts" is ideal for studying human pathologies, patient consultation, or quick reference.
More than 200 images and tables, you may view each image, enlarge it to see every detail.
HC-HealthComm GmbH, Neikesstrasse 3, 66111 Saarbrcken
Telephone-No.:+49 (0)681 9590210-13
E-Mail:
www.hc-healthcomm.com
First edition March 2016. All rights reserved
Anejo Health Communication wwwanejoeu Although the information about - photo 4
Anejo Health Communication www.anejo.eu
Although the information about medication given in this book has been carefully checked, the author and publisher accept no liability for the accuracy of this information.
In every individual case the user must check such information by consulting the relevant literature.
This work is subject to copyright. All rights are reserved, whether the whole or part of the material is concerned, specifically the rights of translation, reprinting, reuse of illustrations, recitation, broadcasting, reproduction on microfilm or in any other way, and storage in data banks.
Table of Contents
Hyperthyroidism e-chart
Hyperthyroidism is a functional disorder in which the thyroid hormone produced by the gland is poured excessively into the blood.
This disorder which encompasses a whole set of anatomopathological structures - photo 5This disorder which encompasses a whole set of anatomopathological structures - photo 6
This disorder, which encompasses a whole set of anatomopathological structures of thyroids, can be divided in hyperthyroidism with ocular disorders (Graves-Basedows disease), and hyperthyroidism without ocular disorders. Some of the most important aspects of hyperthyroidism, also called hyperthyroid syndrome, are depicted and described below.
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Endocrinology Pocket Guide: Full illustrated
Glargine e-chart Insulin glargine is analogous with long-acting human insulin - photo 7
Glargine e-chart
Insulin glargine is analogous with long-acting human insulin. It is obtained through recombinant DNA technology by using a non pathogenic lab strain of Escherichia coli (K12) as the producing organism, which has two molecules of arginine substituted.
12 Endocrinology e-charts More than 200 images and tables - photo 812 Endocrinology e-charts More than 200 images and tables - photo 9
This results into an insu - photo 10This results into an insulin that is soluble in an acidic pH 40 - 50 medium - photo 11This results into an insulin that is soluble in an acidic pH 40 - 50 medium - photo 12This results into an insulin that is soluble in an acidic pH 40 - 50 medium - photo 13This results into an insulin that is soluble in an acidic pH 40 - 50 medium - photo 14
This results into an insulin that is soluble in an acidic pH 4.0 - 5.0 medium, which crystallizes after the injection due to the presence of physiological pH (7.4) in the subcutaneous tissue, this being the reason for the long duration of its action. To stabilize the conditions of solubility in an acidic medium and the precipitation, which leads to tissue degradation, a second change was made by substituting the aspargine at position 21 on the chain (from regular insulin) for a glycine.
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Metabolic Syndrome Pocket Guide: Illustrated
Obesity e-chart It is the excessive rather diffuse or generalized accumulation - photo 15
Obesity e-chart
It is the excessive, rather diffuse or generalized accumulation of fat leading to a significant increase in body weight.
Obesity results from a positive caloric balance due both to excessive food - photo 16Obesity results from a positive caloric balance due both to excessive food - photo 17
Obesity results from a positive caloric balance, due both to excessive food intake and to a reduced caloric output from little physical activity (sedentarism). This disease is very common in those areas in the world with great provisions of food. It is more frequent in groups whose socioeconomic and cultural levels are rather low. Any excessive weight has a negative effect on longevity and on the quality of life.
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Diabetes Pocket Guide 2015: Full illustrated
Signs and Symptoms of Hypothyroidism e-chart The hypothalamus secretes the - photo 18
Signs and Symptoms of Hypothyroidism e-chart
The hypothalamus secretes the thyrotropin-releasing hormone (TRH), which leads the hypophysis to secrete the thyroid-stimulating hormone (TSH). In response to this stimulus (TSH), the thyroid gland releases the stored hormones: triiodothyronine (T3) and thyroxine (T4).
The increase in the levels of T3 and T4 results in a negative feedback both in - photo 19The increase in the levels of T3 and T4 results in a negative feedback both in - photo 20
The increase in the levels of T3 and T4 results in a negative feedback both in - photo 21The increase in the levels of T3 and T4 results in a negative feedback both in - photo 22
The increase in the levels of T3 and T4 results in a negative feedback both in the hypophysis and the hypothalamus, so that the secretion of TRH and TSH becomes inhibited. When the hypothalamus detects a decrease in the thyroid hormones, the cycle starts again.
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